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Kisspeptin-10

low risk

Also: KP-10 · Metastin 45-54

Preliminary Research Only

Kisspeptin-10 is the biologically active C-terminal decapeptide of kisspeptin, retaining full GPR54 receptor activity. It is extensively used in human pharmacology research for HPG axis studies and has potential therapeutic applications in male hypogonadism and fertility.

Molecular Weight
1302.4 g/mol
Formula
C63H83N17O14
Common Dosing
1 nmol/kg IV or subcutaneous (from studies)
Category
research
Last Reviewed
2025-01-15

Reported Benefits

Testosterone Stimulation

Preliminary 15 studies

Subcutaneous kisspeptin-10 reliably stimulates LH and testosterone in hypogonadal men.

Male Fertility

Preliminary 8 studies

Stimulates testicular function in men with certain forms of hypogonadotropic hypogonadism.

Ovulation Induction

Preliminary 6 studies

Small trials suggest potential as ovulation trigger alternative to hCG.

Mechanism of Action

Kisspeptin-10 (the C-terminal decapeptide) activates GPR54 (KISS1R) on hypothalamic GnRH neurons, triggering GnRH pulse release and subsequent LH/FSH-driven gonadal steroidogenesis. The minimal active fragment retains full agonist activity at GPR54.

Key Clinical Studies

Jayasena CN et al. (2011)

crossover · 12 men with T2DM

PubMed →

KP-10 restored LH pulsatility and testosterone in hypogonadal men

Overview

Kisspeptin-10 is the minimal active fragment of kisspeptin, representing the C-terminal 10 amino acids (positions 45-54 of full-length kisspeptin-54). It retains full biological activity at GPR54 receptors while being significantly more synthetically accessible than the full 54-amino acid parent peptide.

Male Hypogonadism Application

The most clinically relevant application of KP-10 research is in male hypogonadism — specifically forms where the HPG axis is intact but not adequately stimulated. In these patients, KP-10 can reliably stimulate LH secretion and testosterone production, raising the prospect of fertility-preserving hypogonadism treatment.

IVF Protocol Alternative

The use of kisspeptin as an ovulation trigger in IVF — as an alternative to hCG — has been studied at Hammersmith Hospital in London. The rationale is that kisspeptin triggers a more physiological LH surge through hypothalamic-pituitary activation rather than direct LH receptor stimulation, potentially reducing ovarian hyperstimulation syndrome risk.

Development Status

Kisspeptin receptor agonists are under active pharmaceutical development by several companies, particularly for infertility indications. KP-10 is the primary research tool compound driving this development pipeline.

Regulatory Status

Research Only

Not FDA-approved; human pharmacology data available

Safety Profile

Side Effects

  • Nausea (injection)
  • Transient flushing

Contraindications

  • Hormone-sensitive cancers

Drug Interactions

  • GnRH analogs
  • Testosterone therapy (monitoring required)

Primary Uses

LH/FSH stimulationMale testosteroneFertilityHypogonadism

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Disclaimer: This information is for educational and research purposes only. Not medical advice. Consult a qualified healthcare provider before using any compound.